南京师范大学C语言程序设计考研近年考试真题笔记2016考研近年考试真题.docx

Hindawi Publishing Corporation
International Journal of Alzheimer’s Disease
Volume 2012, Article ID 630182, 7 pages
doi:
Review Article
Amyloid Beta and Tau Proteins as Therapeutic Targets for
Alzheimer’s Disease Treatment: Rethinking the Current Strategy
Siddhartha Mondragon-Rodr´ ıguez,´ 1, 2 e Perry,3, 4 Xiongwei Zhu,4 andJannicBoehm1
1 Le Groupe de Recherche sur le Syst`eme Nerveux Central, D´epartement de Physiologie, Universit´edeMontr´eal,
Montr´eal, QC, Canada
2 Douglas Mental Health University Institute, McGill University, Department of Psychiatry, Montreal, Quebec, Canada
3 UTSA Neurosciences Institute and Department of Biology, College of Sciences, University of Texas at San Antonio,
San Antonio, TX, USA
4 Department of Pathology, Case Western Reserve University, Cleveland, OH, USA
Correspondence should be addressed to Siddhartha Mondragon-Rodr´ ´ıguez, sidmonrod@
Received 7 October 2011; Accepted 18 November 2011
Academic Editor: Paula Moreira
Copyright © 2012 Siddhartha Mondragon-Rodr´ ´ıguez et al. This is an open access article distributed under the mons
Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is
properly cited.
Alzheimer’s disease (AD) is defined by the concurrence of accumulation of abnormal posed of two proteins:
Amyloid b